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For example, the stimulation of nasal epithelial cells with PM2.5 can increase ROS production through CYP1A1 [49]

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NF-κB enhances cytokine production and immune evasion, while the MAPK axis (extracellular signal-regulated kinases 1 and 2 (ERK1/2), c-Jun N-terminal kinase (JNK, p38) promotes epithelial growth and matrix degradation